Two New Zealanders combine neuroinflammation with the HPA axis to produce a unique explanation for ME/CFS and FM. Neuroinflammation, the hypothalamus and a unifying hypothesis Mackay A, Tate WP. A compromised paraventricular nucleus with a dysfunctional hypothalamus: a novel neuro-inflammatory paradigm for ME/CFS. Int J Immunopathol Pharmacol. 2018:32:1-8. The HPA’s … Continue reading A Novel Neuroinflammatory Paradigm for Chronic Fatigue Syndrome (ME/CFS)